Tag Archives: Thyroid-stimulating hormone

Thyroid Dr Skinner Wins David and Goliath Battle

Thyroid Patient Avocate Dr Gordon Skinner Resumes Normal Practice After Wrongfully Being Accused of Reckless Prescribing

First they ignore you, then they laugh at you, then they fight you,
then you win.”   ~~Mahatma Gandhi

Quote from “What we have learned” STTM

And now for something completely different but absolutely terrific news.

I just read that UK doctor Dr Gordon Skinner has finally been released from his General Medical Council restrictions after 5 years. Is it any wonder doctor’s don’t speak up when those that do are treated in this way.

Why does it take 5 years to work out that thyroid patients are not happy with current medical guidelines on thyroid treatments.

Is it any wonder patients feel they are actually being better treated by natureopaths and “reckless” doctors.

Why don’t legal organisations like this read the forums on the internet? However “subjective” they may be, the sheer volume of complaints must surely be an indication of a discrepency between what is known by doctors and what is known by patients.

Ah well water under the bridge now, but three cheers for Dr Skinner for never giving up on this battle and winning it for all thyroid disorder sufferers as well as for himself.

News Articles

Reckless Mosely Doctor Has Conditions Lifted on His Medical Licence  – After more then five years of restriction, the General Medical Council (GMC) allows Dr Gordon Skinner to resume normal practice.

The Wake You Up Pill – controversial thyroid supplement help tiredness

(Such sensationalist headlines – How about “Effective Thyroid Doctor Wins a David and Goliath Battle About Old School Treatment.”. Or that tacky second headline – how about “Doctor Does His Job Well and Patient Feels Better” Ok, maybe no-one would employ me to write headlines. Who cares, he won!)

Info on Thyroid Treatment Written by Dr Skinner

People Power Will Continue to Work if Patients Sign Skinner’s Thyroid Register

Official transcripts from Dr Skinner’s GMC FTP Hearing and Related Articles

Diagnosis and Management of Hypothyroidism by Dr. G R B Skinner MD, DSc, FRCOG, FRCPath

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TSH Levels Hypothyroidism

TSH Levels and Subclinical Hypothyroidism

Early hypothyroidism may display very mild symptoms or have none at all.

Subclinical hypothyroidism occurs when thyrotropin (TSH) levels are elevated but below the limit representing overt hypothyroidism. The levels of the active hormones,  thyroxine (T4) and triiodothyronine (T3) are normal and will be within the laboratory reference ranges.

Symptoms become more readily apparent in clinical (or overt) hypothyroidism.

In primary hypothyroidism, TSH levels are high and T4 and T3 levels are low. TSH usually increases when T4 and T3 levels drop. TSH prompts the thyroid gland to make more hormone.

The only validated test to diagnose primary hypothyroidism, is a blood test to measure thyroid-stimulating hormone (TSH) and free thyroxine (T4). However it is not totally conclusive, as these levels can also be affected by non-thyroidal illnesses.

High levels of TSH indicate that the thyroid is not producing sufficient levels of thyroid hormones (mainly thyroxine (T4) but also triiodothyronine (T3) in smaller amounts). However, measuring just TSH fails to diagnose secondary and tertiary hypothyroidism. So if the TSH is normal and hypothyroidism is still suspected, further blood testing may be required. Misdiagnosis is common in hypothyroidism, with types of thyroid dysfunction failing to be identified immediately by some laboratory tests. Further tests may be conducted such as…

Measure T3, T4

Free triiodothyronine (fT3)
Free levothyroxine (fT4)
Total T3
Total T4

or other things such as…

Free T3 from 24-hour urine catch
Antithyroid antibodies — for evidence of autoimmune diseases that may be damaging the thyroid gland
Serum cholesterol — which may be elevated in hypothyroidism
Prolactin — as a widely available test of pituitary function
Testing for anemia, including ferritin
Basal body temperature

(S)-Thyroxine_Structural_Formulae L-Thyroxine_...
Image via Wikipedia

There is a range of medical opinion on what agreed upon reference points a patient should be treated with thyroxine, the typical treatment for overt hypothyroidism.

Reference ranges have been debated regarding TSH. The lab that did my results in Australia used 0.4–4.0 mIU/L .  As of 2003, the American Association of Clinical Endocrinologists (ACEE) considers 0.3–3.0 mIU/L within normal range. Previously 0.5–5.0 mIU/L was used, and many doctors (as reported by patients in the health forums) do not treat until the range is over 10.

 

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What is Hypothyroidism?

What is Hypothyroidism?

The pituitary gland and hypothalamus both control the thyroid. When thyroid hormone levels drop, the hypothalamus secretes TSH Releasing Hormone (TRH), which alerts the pituitary to produce thyroid stimulating hormone (TSH).

The major endocrine glands of the body. Pituit...
Image via Wikipedia

The thyroid keeps your metabolism under control with thyroid hormone, which it makes by extracting iodine from the blood.

Every cell in your body depends on the thyroid to manage its metabolism.

Hypothyroidism occurs when the thyroid fails to make enough of these hormones.

When the thyroid gland is unable to produce sufficient amounts of  thyroxine (T4) and tri-iodothyronine (T3) then problems start to occur in all of the body’s systems as the normal bodily functions start to slow down.

All the body hormones work together, so that changes in one effect others. When our body is young this generally works like a well oiled machine. When we pass middle age, the changes affect us more.

This is a possible reason why more women than men appear to be affected by hyperthyroidism especially after they turn 50.

There are three types of hypothyroidism.

The most common is primary hypothyroidism, in which the thyroid gland itself becomes diseased and fails to produce sufficient amounts of the thyroid hormone. The most common forms include Hashimoto’s thyroiditis (an autoimmune disease) and radioiodine therapy for hyperthyroidism.

Secondary hypothyroidism is caused by problems with the pituitary gland and accounts for less than 5-10% of hypothyroidism disorders. It occurs if the pituitary gland does not create enough thyroid-stimulating hormone (TSH) to stimulate the thyroid gland to produce the required amount of thyroxine and triiodothyronine. It is usually caused by damage to the pituitary gland, as by a tumor, radiation, or surgery.

Tertiary hypothyroidism results when the hypothalamus fails to produce sufficient thyrotropin releasing hormone (TRH). TRH prompts the pituitary gland to produce thyroid-stimulating hormone (TSH). Hence tertiary may also be termed hypothalamic-pituitary-axis hypothyroidism. It accounts for less than 5% of hypothyroidism cases.

Patients who have hypothyroidism should exercise caution with certain activities, especially if an activity has a risk of injury (eg, operating presses or heavy equipment, driving, heavy physical labour and contact sports. If their treatment is not yet stabilised and they are having difficulty maintaining concentration in low-stimulus activities, they may have slowed reaction times.

They may also be at risk for ligamental injury, particularly from excessive force across joints due to generalized hypotonia. Hypotonia is a state of low muscle tone (the amount of tension or resistance to movement in a muscle), often involving reduced muscle strength.

Hypothyroidism may be linked to other autoimmune diseases, such as Addisons disease, Chronic Fatigue Syndrome, Diabetes Mellitus, Euthyroid Sick Syndrome, Fibromyalgia, Lupus, Polyglandular Autoimmune Syndrome & Vitiligo.

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